, the bacteria that causes the disease whooping cough
requires various aspects of the immune response for efficient
its host. Using the mouse model, a team of CMIID researchers led by
Eric Harvill and Genetics graduate student Xuqing Zhang determined
that a chemical pathway involved in
inflammatory responses, known as the Interleukin(IL)-1 receptor
pathway, is critical to the survival of mice following B.
"Interleukin-1 Receptor Signaling Is Required To
Overcome the Effects
of Pertussis Toxin and for Efficient Infection or
Against Bordetella pertussis."